Blocking FSH induces thermogenic adipose tissue and reduces body fat.

Document Type

Article

Publication Date

6-1-2017

Institution/Department

MMCRI

Journal Title

Nature.

MeSH Headings

Adipocytes, Adipose Tissue, Adipose Tissue, Beige, Adipose Tissue, White, Adiposity, Animals, Antibodies, Diet, High-Fat, Female, Follicle Stimulating Hormone, beta Subunit, Haploinsufficiency, Male, Mice, Mitochondria, Obesity, Osteoporosis, Ovariectomy, Oxygen Consumption, Receptors, FSH, Thermogenesis, Uncoupling Protein 1

Abstract

Menopause is associated with bone loss and enhanced visceral adiposity. A polyclonal antibody that targets the β-subunit of the pituitary hormone follicle-stimulating hormone (Fsh) increases bone mass in mice. Here, we report that this antibody sharply reduces adipose tissue in wild-type mice, phenocopying genetic haploinsufficiency for the Fsh receptor gene Fshr. The antibody also causes profound beiging, increases cellular mitochondrial density, activates brown adipose tissue and enhances thermogenesis. These actions result from the specific binding of the antibody to the β-subunit of Fsh to block its action. Our studies uncover opportunities for simultaneously treating obesity and osteoporosis.

ISSN

1476-4687

First Page

107

Last Page

112

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